Uric Acid: dietary contribution

Yes, I imagine that the 15%/85% might well be
approximations.
I was concerned mainly from the viewpoint of my
wanting to go on Allopurinol. It is not a drug to be
taken lightly, but some studies show that going off
NSAIDs entirely and taking a course of Allopurinol has
totally ended all gout attacks in patients that
previously had up to three attacks a year. (Yes, study
had relativelyfew participants).
I was mainly concerned with determining my chances of
getting another attack prior to taking the
chemotherapy agent, Allopurinol.
I did go on a low carbohydrate, high protein/fat diet
to lose thirty pound during which I consumed such high
purine items as: liver pate, sausages, turkey.
Combined with the cell turnover from mobilizing stored
fat, my high purine diet may have contributed to this
initial attack of gout which is still lingering.
IF I do not repeat the poor dietary selection and the
weight loss program, I might be able to avoid future
attacks. This ofcourse would make Allopurinol a very
poor choice to elect at this point in time.
It seems that if this "ten to fifteen" percent dietary
is a reasonable estimate, that my dietary selections
might have been a "tipping point" for the attack even
though the weight loss was six months before symptom
onset.

Comments

1 Responses to Uric Acid: dietary contribution

  1. gil_900 on 2006-12-21 13:14:59.995184

    "my wanting to go on Allopurinol. It is not a drug to be taken
    lightly, but some studies show that going off NSAIDs entirely and
    taking a course of Allopurinol has totally ended all gout attacks in
    patients that previously had up to three attacks a year. (Yes, study
    had relativelyfew participants)."
    Your research is incomplete. Allopurinol works for everyone who can
    tolerate it, all the time, in every case. There are literally
    thousands of studies where this has been reported to be true. There
    are no cases reported, at least that I could find, where it did not
    work if the patient could tolerate the drug.
    "chemotherapy agent, Allopurinol."
    Yes, allopurinol is used in chemotherapy. However, it is not used as
    a primary agent against cancer. It is used in conjunction with
    reagents and radiation that kill the cancer.
    Why is it used in chemotherapy?
    The nobel prize was awarded for discovering its function by the way,
    and what it does in chemotherapy is exactly what it does for us with
    gout. It stops the production of uric acid.
    So why would cancer patients need that?
    When you kill a cancer, or large parts of a cancer, you end up
    putting an overload on the bodies internal digestive system and you
    overload that system with DNA and RNA from dead cells that were
    killed by the chemo drug or that were killed by the radiation. These
    excess cells end up flooding the system with too much purene for the
    already weakened system to handle and uric acid poisoning often
    results. Gout, kidney stones, kidney blockage etc. are among the side
    effects of the chemotherapy that results from killing a large number
    of cells in the body quickly. Allopurinol is used to block the
    pathway to uric acid, leaving the end products of cell metabolism in
    a more water soluble form that is more easily excreted by the kidney.
    I don't know what country you are from but you suggested that
    physicians are reluctant to perscribe Allopurinol. Clearly you are
    not in the USA because this is not the case here. It is a very low
    cost and relatively easy to get drug. It is safe for most people
    although ther are side effects, mostly an alergic reaction that is
    most likely to occure when you first start taking the drug. Because
    of this possible allergic reaction, some physicians will start you
    off low and build up your dosage and some will just jump in with much
    larger dosages and then reduce the dose when the gout is gone. Mine
    started me on 100 mg a day and I have increased my own dosage now up
    to 300 a day. The symptoms did not seem to go away at all on 100 mg a
    day and now on 300 they seem to be subsiding.
    It seems more logical to me that if you had simply increased your
    rate of uric acid removal-if you had drunk more water-you might also
    have avoided the attack...

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